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LEG 500 Strayer University The Value of Fair Treatment in the Workplace Response

LEG 500 Strayer University The Value of Fair Treatment in the Workplace Response.

The Value of Fair Treatment in the WorkplaceYou have been hired as a management consultant by a large company to examine the company’s business decisions regarding employee protections. The applicable laws are federal anti-discrimination laws, federal health and safety laws, and employer firing practices related to the employment-at-will (EAW) doctrine.In response to the three questions below, write a 4–6 page paper in which you do the following:Analyze, identify, and explain recent legislation, within the last 10 years, that helps to protect employees from discrimination in the workplace. Provide at least two federal legislative protections. Provide some insight when the federal legislation conflicts with the state. Explain the EAW doctrine and all exceptions to the doctrine. Look at the scenarios below and determine whether the decision to fire the employee is a smart one. Identify why or why not, and determine all the possible exceptions per the EAW doctrine that are, might be, or could be applicable if the employee sues for wrongful termination.Brenda, a manager, started a blog on the company website for employee grievances. She noticed that a worker was protesting that no Asian American employees had gotten a raise in two years at the company. The worker also criticized how much the CEO made and how the CEO was “out of touch.” Brenda reprimanded the worker. The next day, the worker talked to fellow co-workers about forming a union. Brenda subsequently fired the worker.Jason, a department supervisor, requests approval to fire his secretary, Alice. Alice, a devout Christian, has been putting Right-to-Life flyers in the employee breakroom. Alice is also taking time out to pray each day during the busiest time of the morning.Brian, the head of the accounting department, refused to sign Lori’s leave request for jury duty. Lori is a tax attorney in his department. Brian wants to fire Lori for being absent without permission during their busiest time—tax season.Peter has worked for the company for one year. He has a rare form of liver disease and works with chemicals that make his condition worse. Peter does not want to stop working, but his boss is not happy with his performance and wants to let him go.Determine the federal law regarding undocumented workers and whether they are eligible for state workers’ compensation in the United States. Advocate for or against this practice and substantiate your response with research to support your position.Use the Strayer Library to conduct your research. Include at least three quality references. The textbook for this class is a required source for this assignment.Note: Wikipedia and similar websites do not count as quality references.Your assignment must follow these formatting requirements:This course requires the use of Strayer Writing Standards. For assistance and information, please refer to the Strayer Writing Standards link in the left-hand menu of your course.Typed, double-spaced, using Times New Roman font (size 12), with 1-inch margins on all sides.Include a cover page containing the title of the assignment, the student’s name, the professor’s name, the course title, and the date. The cover page and the reference page are not included in the required assignment page length.The specific course learning outcome associated with this assignment is as follows:Evaluate federal law relative to workplace discrimination, employment-at-will, and worker’s compensation eligibility.
LEG 500 Strayer University The Value of Fair Treatment in the Workplace Response

DB2 responses for 2 of my classmates “Healthcare Emergency Mgmt “. I don’t know how to handle this Management question and need guidance.

I have 2 of my classmates posts. I need you to respond to each one separately. Also, two sources at least for each one of them. Don’t write about how good their posts or how bad. All you need to do is to choose one point of the post and explore it a little bit with one source support for each response. The paper should be APA style.
– pleas use proper APA6 references format and in-line citation.
– Minimum words for every respond 300
-Minimum refernces 2
All the details in the attached below
DB2 responses for 2 of my classmates “Healthcare Emergency Mgmt “

The Irish Influences in the Work of W. B. Yeats

The Irish Influences in the Work of W. B. Yeats. Can you help me understand this English question?

Yeats Five: The Final Draftplease add more words (1800-2700)

The research paper does not fulfill the requirements of the assignment because it is too short. – I will not accept papers that do not fulfill the minimum assignment requirements.
The research paper is 1,800–2,700 words and MLA 8 FORMATTED. WC 1146– TOO SHORT – Stylistically, more detail is needed to create a research paper. Keep in mind that research papers are not only content and formatting, but length as well. A word count of less than 1500 words is stylistically an essay, not a research paper. Content less than 1500 words (an essay) will not count as fulfilling the minimum requirements of this research paper assignment and will be returned until the length of the paper is appropriate for the assignment.
The Irish Influences in the Work of W. B. Yeats

Production of Mutant Based Epsilon Toxin (ETK) Vaccines

help writing Production of Mutant Based Epsilon Toxin (ETK) Vaccines. Critique of Li et al. Article: A low-toxic site-directed mutant of Clostridium perfringens É›-toxin as a potential candidate vaccine against enterotoxemia Donnelle Phillips Vaccines improve the immune system of an animal or human by stimulating the production of antibodies to fight and combat bacteria, protozoan or viruses effectively creating immunity against disease (Salisbury 1997). Vaccines commonly contain weakened or dead strains of a virus, bacteria or protozoan which elicits the body’s immune system upon recognition to respond by producing antibodies to fight against the invading antigen. In effect, the vaccine introduces the disease or its causative agent into the body to gain a defence response; as a result if the disease enters the body in the future the body can produce the requisite antibodies to fight the disease faster, as it remembers measures taken to fight against the disease previously. Consequently, the time taken to recognize the disease, provide active measure against the disease and minimize any possible signs or symptoms associated with the causative virus, bacteria or protozoan. Building on this function of vaccines this essay critiques the study and subsequent paper of Li et. al. investigation into the production of mutant based epsilon toxin (ETK) vaccines which can provide protection against intoxication. Costridium perfringes a Gram positive spore forming bacterium, is ubiquitously found in the environment and the intestines of humans and animals. Furthermore, it is commonly found in raw meat and poultry and is also linked to foodborne illnesses and food poisoning (Centers for Disease Control and Prevention 2014). Presently, there are five known strains of C. perfringes which produce a minimum of twelve known protein toxins (The Centre for Food Security and Public Health 2004). To this end, the epsilon toxin (ETX) is produced by the type B and D strains (The Centre for Food Security and Public Health 2004). Moreover, ETX is a pore forming protein which damages cell walls leading to potassium and fluid leakage from cells and it increases the permeability of the cell membrane resulting in ion imbalance; as such ETX has been suggested as a possible biological weapon (Stiles, et al. 2013). Arizona Department of Health (2004) speculates that in the event of a biological attack the epsilon toxin can be spread through food, water or by aerosolization. While all five strains have the capacity to infect wounds in any species; type B and type D have the ability to cause severe enteritis, in young foals, lambs and piglets, and enterotoxemia in young calves, lambs, goats, sheep and foals just to mention a few (Arizona Department of Health Services 2004). The toxin is also known to cause pulmonary edema and neurological symptoms including recumbence hyperesthesia, convulsions, paddling, dyspnea and loss of conciseness when intravenously injected into animals. They are few reported cases on the toxicity of ETX in humans as the type B and D strains rarely infect humans. However, evidence suggest that it can affect the G-402 and ACHN cell lines of the human kidney highlighting its possible toxicity to humans (Li, et al. 2013). The epsilon toxin is secreted as an inactive prototoxin from the cytosol into the environment as a result of a 32 amino acid residue leader sequence. It is then activated by extracellular serine type proteases of the host such as trypsin or chymotrypsin; which subsequently results in the loss 10-13 amino terminal and 22 or 29 carboxy terminal residues depending on the protease used (Stiles, et al. 2013). Moreover, maximal activation results in the loss of 13N-terminal residues and 29 C-terminal residues producing the third most potent toxin of the clostridia class, loss of these two terminals results in the formation of a more acidic protein which possibly favours more productive receptor interactions (Hunter, et al. 1992). Bokori-Brown et al. (2011) states that the prototoxin can also become activated by C. perfringens λ – protease. When activated the toxin becomes relatively resistant to proteases in the gastrointestinal tracts of mammals (Stiles, et al. 2013).The effects of the toxin on the cell membranes lead to cell dysfunction, cell death and suffering in the host which can sometimes be fatal. Proteolysis induces a conformational change in the toxin facilitating homo-oligomerization of the activated toxin on the external surface of the cell (Stiles, et al. 2013). After binding to a cell ETX acts quickly. Two groups of pore forming toxins exist, to this effect ETX is a beta pore forming toxin as it forms a beta-hairpin structure to facilitate membrane insertion (Stiles, et al. 2013). Moreover, the toxin has three domains with implications in the function of the toxin. The first domain, domain I-amino terminus, functions in receptor binding, the central domain or domain II functions in membrane insertion and channel formation (Stiles, et al. 2013). The final domain, domain III located at the carboxy terminus functions in proteolysis activation and act as monomer to monomer interaction sites. Subsequent to the loss of the C terminal peptide from the protoxin monomer-monomer interactions leads to homo-heptamer formation. ETX cause pore formation in cell membranes by detergent-resistant, cholesterol-rich membrane domains that promote aggregation of toxin monomers in homo-heptamers (Stiles, et al. 2013). Evidence from research highlights ETX forms transmembrane pores less than two nanometre in diameter that facilitates the passage of molecule 1kDa in size, increase intracellular chloride and sodium levels and decreased intracellular potassium levels. Furthermore, secondary effects of the toxin include cytoskeletal dysfunction which affects the integrity of cell monolayers (Stiles, et al. 2013). The general layout and presentation of the paper was good and easy for readers to follow. However, the materials and methods should have occurred before the results and discussion to familiarise the readers with the methods used in the experiment. In doing this readers would have an idea of the type of results to be presented or expected. Furthermore, if presented earlier the procedure could be borne in mind in order to help readers understand the information presented in the results and discussion sections and the relevance of the information to the aim of the research. Additionally, if the methodology is placed earlier in the paper it helps readers to determine the extent of the internal validity of the study, as readers can determine for themselves the extent to which results can be interpreted accurately and with confidence with regards to the methods used to obtain the presented findings. Reliability, a necessary condition for validity, is related to consistency of results and the extent to which the research can be independently replicated by other researchers (Leacock, Warrican and Rose 2009). Based on the information provided the study is reliable and can be replicated independently by researchers with the relevant resources. In addition the use of graphs, tables and photographs of the gels used in the experiment are good means of presenting information. To this end, it presents data in a concise format allowing readers to easily identify pertinent information and reduces the possibility of readers being overwhelmed if this information was to be fully present using only text. It is also important to note that the use of the photos form the SDS-PAGE and the immunoblotting aids in reader visualisation and acts as a point of reference for readers when examining the results and discussion. The introduction presents a good base for the readers but would have benefited from more in depth explanation on the mode of action and structure of ETX and more background information on mutant based vaccinations. Also a proper comparison of the proposed mutant based vaccine and how it would essentially function in contrast to the existing vaccine which only protects against enterotoxemia caused by the disease would have increased the reader’s knowledge about the topic and reinforce the importance of find a viable vaccine with low toxicity. It is important to note that the introduction provided supportive information from previous research about the amino acid residues present in the structure of ETX and their function. By extension the introduction also highlights the chosen sites for mutation in the toxin based and evidence also presented in the paper. A brief synopsis of the methods used with reference to article presented by Li et al. are as follows; sited directed mutagenesis of ETX in the pTIG-trx plasmid, with the obtained sequence being confirmed using nucleotide sequence analysis. Following this the pTIG-mETXs were transformed into competent E. coli for expression which was also driven with a T7 promoter. The bacteria was then collected and purified through a combination of centrifugation, buffer washes, resuspension, sonification, chelation and elution. The purified protein were subsequently analysed using SDS-PAGE, with the highly purified proteins undergoing dialysis and concentration. The next step employed ELISA to identify the antigenicity of the mETXs and rETX using anti-rETX monoclonal antibodies. After electrophoresis the purified mETX protein were transfer from the SDS-PAGE gel using the western blot technique. The ELISA and western blot steps served to illicit the antigenicity of the mETX which both involving the use of goat anti-mouse IgG during incubation periods. A cell culture and cytotoxicity assay was conducted to determine rETX activity by defining its effects on MDCK cells. Three dimensional structure of two mETXs, mETXF199E and mETXH106P, were generated for structural analysis. The two mentioned not-toxic mETXs were used to vaccinate six week old female mice with the same dosage of antigen being given on days 17 and 38 a week later the mice were injected with active recombinant mETX and observed for 72hours. The following step involved the measurement of sera antibody titers. The final step of the experiment tested mice for passive protection against rETX. The use of ELISA is a good means of detecting the presence of ETX and is one of the assays highlighted by the Arizona Department of Health Services for detection of the toxin due to its high detection rates (Stiles, et al. 2013). ELISA combines the specificity of antibodies with the sensitivity of simple enzyme assay by coupling the two, in the experiment it was us to detect the presence of antibodies which recognize ETX (Sino Biological Inc 2014). Additionally, the use of coloured tags indicate a positive reaction when the substrate interacts with the enzyme which also gives visual confirmation of the presence of the antigen or antibody which would have useful implications for this study. Furthermore, it is effective as it produces relatively fast results, which is especially relevant as samples can degrade over time, deterioration may also have implications for the levels of antibodies present. Liquid chromatography-mass spectrometry uses inmmunoaffinity beads to concentrate ETX or prototoxin from a complex matrix and is also a method commonly used to determine the presence of ETX and may be given consideration if the study is replicated (Stiles, et al. 2013). While ELISA and mass spectrometry are commonly used in the detection they do not determine if the toxin is biologically active. The use of titration was effective as it determined the amount and levels of antibodies required to neutralize the toxin. Random assignment to treatment groups is good as it gives each mouse the same change of being assigned to a group and reduces possibility of bias. However, as only three mice there were used in each group there are possible implications for the analysis of the results, as for experimental research the minimum recommended number is 15 participants for each group, as a sufficiently large sample size is required to produce results among variables that are significantly different (Leacock, Warrican and Rose 2009). A bigger sample size may also be effective in giving better credence to the obtained results for each treatment. The use of only female rats can have implications for the generalization of the results and by extension the validity of the study. Furthermore, all of the mice used in the experiment were six weeks in age this does not provide variation and as such does not account for the possible effects age can have on the function of the vaccine and results of the rETX challenge. Validity applies to the generalization of research and the vaccines is aimed for use in animals and humans and these populations contain both males and females, thus the use of one sex in the experiment have implications for generalization of results. The study may have also benefited from longer observation period after the mice were injected with the rETX. Despite the potential market for the vaccine including humans the research was conducted using mice due to ethical considerations. Additionally, the creation of pharmaceutical therapies require several animal experiments to be conducted using the treatment under before the ability for a clinical study using humans and the vaccine being offered to the public. The result section has sub headings corresponding to the methods used which aids in easy identification for readers. Some of the significant findings of the research include the yield of six mETXs and up to 98% purity was obtained after purification measures. Additionally, the results showed that the toxin mutants retained the same antigenicity as the rETX. Four of the toxin mutants showed decreased cytotoxicity, while mETXS111Y and mETXS111YF199E showed a slight decrease in toxicity and higher toxicity when compared to rETX thus the researchers excluded the latter two from further analysis. Also of importance is immunization and the subsequent ELISA results of the anti-mETX antibody titres highlighted mETXH106P and mETXF199E showed similar titres and there was no significant difference between the two; but antibody titers also increased after booster immunization. The most significant results demonstrated that mice when challenged with active rETX with dosages up to 100 x LD50 survived. In contrast all with dosage of 500 x LD50 or 1000 x LD50 died which have implications for use of the vaccine and provides grounds for further research to obtain vaccines which have a better response to higher dosage of rETX. Investigation into passive protection showed anti-mETXH106P or anit-mETXF199E can completely neutralize a 10 x LD50 dose of activated rETX. The discussion highlighted supporting facts from various sources but there was limited discussion to certain findings of the paper. A possible suggestion is that the authors provide a more in depth discussion about their findings instead of a synopsis. The discussion may have also benefited from a comparison of the mutant based vaccine with the effectiveness of current ETX related treatments. Furthermore in depth explanations of the findings would help readers understand the significance of the findings and implication for future production and use of the tested vaccine. A significant finding highlighted in the discussion is that modifications of various amino acid residues result in varying cytotoxicity of the ETX. To this end, the researchers should have discussed why all of the obtained mETX were not tested in the immunization phase. A recommendation is the use of all of the obtained mETXs in the vaccination section of the experiment to determine the possibility of its use as a vaccine to provide immunity against the epsilon toxin. The study did record some success in meeting its aims as two of the mETXs, mETXsH106P and mETXF199E as possible candidates for vaccines against the toxin as they showed strong immunogenicity and safety. However, mETXF199E still has toxicity, as a consequence the researchers have made a possible suggestion to improve the vaccination and lower toxicity. In concluding this article has led to the advancement of the body of knowledge pertaining to protection against ETX. The information from the study despite some limitations is a good read for persons interested in the topic. To this effect, it also provides a theoretical and empirical basis for further study about therapeutic measures to combat the epsilon toxin. It is important to note that it provides a preventative method against the toxin where as other treatments tend to target the ETX related diseases and related symptoms of the toxicity of the toxin, which some treatments delaying the onset of the toxin’s effects or delaying or preventing death (The Centre for Food Security and Public Health 2004). References Arizona Department of Health Services. 2004. Epsilon Toxin of Clostridium perfringens Bioterrorism Agent Profiles for Health Care Workers. August. Accessed March 20, 2015. Bokori-Brown, M, C G Savva, S P Ferandes da Costa, C E Naylor, A K Basak, and R W Titball. 2011. “Molecular basis of toxicity of Clostridium perfringens epsilon toxin.” The FEBS Journal 278 (23): 4589-4601. Centers for Disease Control and Prevention. 2014. CDC- Clostridium perfrines – Food Safety. January 29. Accessed March 20, 2015. Hunter, S E, I N Clarke, D C Kelly, and R W Titball. 1992. “Cloning and nucleotide sequencing of the Clostridium perfringens epsilon-toxin gene and its expression in Escherichia coli.” Infection and Immunity 60 (1): 102-110. Leacock, C J, S J Warrican, and G Rose. 2009. Research Methods for Inexperienced Researchers. Kingston: Ian Randle Publisher. Li, Qing, Wenwen Xin, Shan Gao, Lin Kang, and Jinglin Wang. 2013. “A low-toxic site-directed mutant of Clostridium perfringens É›-toxin as a potential candidate vaccine against enterotoxemia.” Human VaccinesProduction of Mutant Based Epsilon Toxin (ETK) Vaccines

Species-area Relationship of Herpetofaunas in West Indies

De Gao 1. Introduction 1.1. Biodiversity and crisis on West Indies Oceanic islands, due to their discrete geographic isolation, differ distinctly from terrestrial ecosystem, offer great opportunities for species conservation, and are regarded as the natural laboratory for the study of biogeography and evolutionary ecology (Adsersen, 1995; Crawford and Stuessy, 1997; Ricklefs and Bermingham, 2008). Island habitat heterogeneity and the gene flow barrier between the island and its neighboring islands or land are the most important driving forces leading to new species differentiation and formation (Cherry et al. 2002). The colonized species from continents or nearby islands are then subjected to unique evolutionary processes, for example, geographic isolation, character displacement, and adaptive radiation. The accumulated mutations gradually and ultimately make colonized species into new indigenous species that are genetically different from the original one. One of the most distinctive features of the biota on oceanic islands is the large number of endemics occurring in small areas (Francisco-Ortega et al. 2000). West Indies was identified as a biodiversity hotspot by Myers et al. (2000). A significant percentage of the Earth’s known terrestrial biota is distributed on islands of the West Indies, many of these species are endemic to the region, to individual islands, and even to isolated areas within some islands (Hedges, 2001). But a large majority of the vast complex of islands in West Indies is changing rapidly, resulting in dramatic alterations of habitats, and nearly all of them are mediated by human activities (Powell and Henderson, 2012). Small population size, gene drift, isolated gene flow, and the complexity of island environment have already made the endemic herpetofaunal species easy to be endangered or extinct, human disturbances undoubtedly worsen the situation. Therefore, critical conservation concerns in West Indian region are increasingly important and required. 1.2. Species-area relationship (SAR) The species-area relationship is one of the most fundamental patterns in macroecology and an important tool in assessing species diversity, extinction rates and species hotspots (Tjørve, 2009). According to the equilibrium theory (MacArthur and Wilson, 1967), the relationship between immigration and the extinction of species to island depends on the island size and its distance to mainland. MacArthur-Wilson’s theory provided impetus for numerous studies on species-area relationships (SARs) that have provided biological interpretations for several taxa and have suggested factors and mechanisms that can determine species richness (Simaiakis et al. 2012). And the theory has an enormous influence on perspectives of species conservation and nature reserves design. The main objective of this chapter is to discuss SARs for herpetofaunas in West Indies regions, with the aim of discover which factors may determine their distribution over the whole range of island sizes. We choose to fit some candidate models including the power model and some sigmoid models to see whether a J-shaped part exists at the lower end of the curve. Small-island effect (SIE) holds that, on islands smaller than a certain critical minimum area, one cannot observe an increase in species number with increasing area (Simaiakis et al. 2012). We choose to fit candidate models including the power model and some sigmoid models to see whether a J-shaped part exists at the lower end of the curve. If the best fitted model has a sigmoid shape, a J-shaped part will exist at the lower end, and then we accept SIE. If the best fitted model has a convex shape, a J-shaped part will not exist at the lower end, and then we deny SIE. There are also other reasons why it is so important to know the shape of species-area curve. Based on the curve shape, we could estimate total species diversity from limited numbers of samples, or estimate species extinction as a result of habitat loss and fragmentation (Tjørve, 2003). Candidate species-area models are proposed from two sources. The first source is species-area literature which has traditionally fit data with power or exponential models, the second source involves a broader search of the literature for models that increased monotonically to an asymptote (Flather, 1996). Although the shape of the curve has been extensively debated over the years, power and exponential curves are the most commonly applied and best known ones, lately, an increasing number of authors have argued some SARs are perhaps best represented by functions that are sigmoid in arithmetic space and there are also a number of recent studies where sigmoid models have been fitted to empirical data (Tjørve, 2003). Some sigmoid models behave similar with others, for instance, the Extreme value function behaves quite similar to the Logistic and Gompertz models; The Cumulative beta-P distribution behaves similarly to the Weibull distribution (Tjørve, 2003). 2. Aims In this study, we are trying to solve 3 questions: Question 1: What is the species-area relationship (SAR) for herpetofaunas in West Indies? Is the SAR same for native and invasive species? And is it the same among each animal group classification under amphibian and reptile? Question 2: Due to human’s help, invasive species are supposed to have a higher dispersal ability as compared to the native. So, in the linear form of the power model (LogS/LogA) is there a higher z-value for the invasive species than the native species? Question 3: What geographic factors may determine species richness over the whole range of island sizes? Are these factors also applicable to other group of islands that located in similar latitude areas to determine their species richness? 3. Material and Methods Data on species occurrences originate from Powell and Henderson (2012). Multiple basemaps in ArcMap 10 and ArcGlobe 10 will be used to digitize islands map. We use different scales to digitize islands with various areas. Fractal Analysis will be used to estimate the perimeters at a uniform scale (Farina, 2007). For comparisons of the fit of different models, the Akaike’s information criterion (AIC) was applied (Burnham and Anderson, 2002). Model selection analyses will run using “mmSAR” package (Guilhaumon et al. 2010) for the R statistical and programming environment (R Development Core Team, 2011). Aim 1: Model selection we use the 8 most general models composed in “mmSAR” package (Guilhaumon et al. 2010) to fit the data, which include 5 convex models (Power, Exponential, Negative exponential, Monod, Rational function) and 3 sigmoid models (Logistic, Lomolino, Cumulative Weibull). Aim 2: LogS/LogA linear regression We analyze the linear function of the LogS/LogA and compare parameter values with vertebrates from global oceanic islands (Triantis et al. 2012). Aim 3: Find out the geographic factors affect species richness/Steps Initially selected variables are X1 Distance to mainland X2 Latitude of island centroid X3 Longitude of island centroid X4 Island No. within 0.1km X5 Island No. within 0.1-1km X6 Island No. within 1-5km X7 Island No. within 5-10km X8 Islands Area within 0.1km X9 Islands Area within 0.1-1km X10 Islands Area within 1-5km X11 Islands Area within 5-10km X12 Area of the island X13 Perimeter of the island X14 Perimeter/Area X15 Percentage of vegetation X16 Percentage of human area X17 Rivers within the island X18 Invasive spp. within the island X19 Percentage of invasive spp. X1 and X3 are used to examine species dispersal from mainland; X2 is used to examine the nearness to equator; X4, X5, X6, X7 are used to examine stepping stones in each loop circle; X8, X9, X10, X11 are used to examine total area of stepping stones in each loop circle; X12 is used to examine area effect; X13 and X14 are used to examine shape (irregularity) of island; X15, X17 are used to examine habitat quality; X16, X18, X19 are used to examine human disturbance and invasive species. Principal Component Analysis and Hierarchical Partitioning Analysis will be made based on the residuals distribution of LogS/LogA linear regression to examine which geographic factors are the significant contributions of species richness in West Indies.

Aspen University Nursing Practice Specialty Area Discussion Post

Aspen University Nursing Practice Specialty Area Discussion Post.

I’m working on a nursing question and need a sample draft to help me learn.

Part IDiscussion Question:Think about your unique nursing practice specialty area and the population you serve. Is there a clear connection to practice and theory in your specific nursing specialty area? In this discussion post, you will describe the relationship between theory and nursing practice and how you could implement theory into nursing practice.Describe why nursing theory is important to today’s nursing practice.How would you select and introduce a nursing theory or model into your nursing practice?Your initial posting should be at least 400 words in length and utilize at least one scholarly source other than the textbook. Please reply to at least two classmates. Replies to classmates should be at least 200 words in length.Part IIAssignment:Nightingale Case StudyAssignment Description:Write a 1000-1500-word APA paper addressing each of the following points. Be sure to completely answer all the questions for each bullet point. Separate each section in your paper with a clear heading that allows your professor to know which bullet you are addressing in that section of your paper. Support your ideas with at least three (3) outside sources and the textbook using citations in your essay. Make sure to cite using the APA writing style for the essay. The cover page and reference page in correct APA do not count towards the minimum word amount. Review the rubric criteria for this assignment.Case Study: Mrs. Adams, a 68-year-old widow who was referred to case management upon discharge from the hospital based on her physician’s recommendation that she is not able to care for herself independently. Her diagnosis is diabetes, hypertension, and breast cancer. She is 5 days’ post-op from a right sided mastectomy. Mrs. Adams apartment is located in a low-income area of the city where crime is prevalent. Upon assessment by the Community Health Nurse, Mrs. Adams apartment was in disorder with minimal airflow or light. Her clothes appeared unchanged and she had no food in the apartment. The small apartment also housed 3 cats and a small dog who Mrs. Adams considers family since the death of her husband 1 year ago. Mrs. Adams complains of pain and draining from her surgical site and a broken air conditioner. Using Nightingale’s Environmental Theory, what actions would the nurse take upon the first assessment? What are the five essential components?What would be an appropriate plan of care be for Mrs. Adams? How would you prioritize the care plan?Apply Nightingale’s Environmental Theory to an area of your nursing practice? What patient-population would benefit from this approach? Support this practice change with at least one evidenced-based article. Assignment Expectations:Length: 1000 – 1500 wordsStructure: Include a title page and reference page in APA format. These do not count towards the minimum word count for this assignment. Your essay must include an introduction and a conclusion.References: Use appropriate APA style in-text citations and references for all resources utilized to answer the questions. A minimum of three (3) outside scholarly sources and the textbook are required for this assignment.Rubric: This assignment uses a rubric for scoring.
Aspen University Nursing Practice Specialty Area Discussion Post

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