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ENG112 Pitt Community College The Life of Martin Luther King Jr Paper

ENG112 Pitt Community College The Life of Martin Luther King Jr Paper.

You will have one body paragraph for early life (before college-age); one body paragraph for college
or training (if applicable) and/or early career; one body paragraph for their main career (where most
people’s best accomplishments will be); and one body paragraph for their later life, death, and legacy.
You will also have an introductory paragraph (concluding with a thesis statement that informs the
reader of the four main sections listed above to be covered) and a conclusion paragraph.
This is an explanatory paper, meaning you are simply describing/explaining the life of your subject to
the reader. Your tone should be dispassionate; you are just a reporter. Do not inject personal spins or
feelings anywhere in this paper. Your duty here is to inform, not to argue a point. Do not use the first
person (I/me/my statements) at all (unless quoting, of course). Place yourself “in the background” in
this paper. For more information on explanatory writing, see Everyone’s An Author (pp. 252-286) and
They Say/ISay (pp. 19-51).
You will use a minimum of four sources, though you may use more. Please consider not just the public
internet and print sources, but also sources from our library’s databases (links to selected databases on
Moodle). Remember to evaluate all sources for appropriate authorship, sponsorship, and currency (see
Evaluating Sources handout on Moodle). You must cite something in-text at least once in each body
paragraph (you may cite in the intro and conclusion, but it’s not required). Don’t forget to include a
properly formatted Works Cited page, and make sure every source on your list is cited somewhere in
the paper at least once. For more information on in-text citations and MLA Works Cited pages, see the
Purdue OWL website or the Little Seagull Handbook.
Remember to adhere to MLA format (see Purdue OWL website or the Little Seagull Handbook).
Follow the rubric posted on Moodle carefully to make sure you adhere to all the requirements of the
assignment. Here are a few helpful hints.
• Cite not only after direct quotations, but also after paraphrased/summarized information. When
in doubt, do an in-text citation. It’s better to cite too much than too little.
• Avoid making an announcement (e.g. “This paper will be about…” or “In this paper, we will be
discussing…” etc.).
• Avoid using “you” whenever possible. It can upset the tone of your paper.
• Be mindful of tense. Stay in a consistent tense (in this case, past) unless you need to shift to
another tense. Then always be sure to shift back to the original tense.
• Avoid referring to your subject by their first name. It looks unprofessional. When writing about
people who are revered and respected, it’s much better to refer to them by their last name.
• Keep your tone firm and assertive. Make sure the reader feels you know what you’re talking
about. From their perspective, you’re the expert.
• Be creative when searching for sources. Think about video documentaries, audio interviews
with your subject, etc. Those can make excellent sources.
ENG112 Pitt Community College The Life of Martin Luther King Jr Paper

CU Explanation on The Different Sources of Funding the Company Can Have and Their Advantages and Disadvantages Discuss.

The detail can check on the DOCXAssignment TitleManagement ReportAssessment Learning Outcomes This assignment is designed to assess all learning outcomes: MLO1 to MLO6Details of the taskCASE STUDY: Jackson LimitedJackson Limited is a UK subsidiary of Adams Group whose head office is in Kenya. The company has been operating in the UK for the past 20 years, providing a range of services to SME’s, namely investment property funds. Over the past two decades, Jackson has been a profit-making firm, retaining its previous clients, in addition to capturing an increasing share of the market. However, the finance director of Jackson has recently got in touch with your professional consulting firm and has engaged your firm to provide them with an explanation of the cash flow problem that Jackson Limited had been facing. The company is also dependent on the parent based in Kenya for and when required.Following recent meetings of the senior managements in London and Kenya, it has been agreed that Jackson Limited should do expand the business further and raise the required capital in the UK, or perhaps in Europe, so as not to depend so much on cash coming from the parent company all the time. Consequently, the management of Jackson is considering the followings:New information systemJackson’s current system that is used to provide specialised services to its clients’ needs to be updated. The company is considering an investment in a more modern system and two possible alternatives have been proposed as outlined below.Golden packageGolden PackageDraft figures£’000Year012345New system’s costs11,500Cumulative Working Capital800500700200600Sales Revenue1,4005,1006,5005,0003,500Less:Segment A(540)(600)(950)(980)(1,200)Segment B(1,150)(1,650)(1,800)(2,200)(1,800)Overheads(200)(250)(350)(320)(310)All of the above estimates have been prepared in terms of present day cost and prices. Assume that cash flows arise at the end of each period. In additionRevenues are expected to rise by 4% in price terms per year from year 1 (start of year 2) the budget estimated selling price at start was £120.Overheads and working capital are expected to rise by 4% per year from year 1 (start of year 1)The cost of Segment A and Segment B are expected to rise in line with inflation of 4% per year from the beginning of year 1The working capital is cumulative and will be recouped at the end of year 5The cost of System Experts (SE), who have come from the Kenya have not been taken into consideration in the forecast and are as follows:System Expert 1 (SE1): Will be paid £130 per hour and expected number of hours for SE1 are 1,300hrs. The rate paid is expected to rise in line with inflation at 4% per year from year 2 and the number of hours is expected to reduce by 3% per year, every year from year 2 onwards.System Expert 2 (SE 2): Will be paid £125 per hour and expected number of hours for SE2 are 1,400hrs. The rate paid is expected to go up in line with inflation at 4% per year from year 2 and the number of hours is expected to reduce by 4% per year, every year from year 2 onwards.If Jackson Limited invests in Golden Package, then the discount rate that would be required to assess the NPV would be 10%. The table above shows the estimated outgoings and inflows for the project. Premier CentreThe Sales Manager of Jackson has just informed your company that they plan to open a centre in the UK, with a possible start date of business on 1 August 2020. You have also been informed that to start with, the company will only sell 2 new types of service packages, that of Basic Level (BL) and the Professional Level (PL). The intentions here is to test the market and check whether they can achieve break-even in the same period. A recent market research has suggested that these two are the most popular systems and will be offered at £350 for BL and £450 for PL.You have also been provided with the following information regarding the costs and estimated sales for the period mentioned above.Jackson Limited intends to put in £6,000 as start-up capital and plan to sell a total of 1,200 (combined) of BL and PL for the same period. They are not sure which of the two types of service packages will produce the most profits for Jackson.Total budgeted sales for each month are as follows: August 400, September 400 and October 400, of which 40% of each month will be for BL. You will be required to assess the best product combination of sales for the period.To help with the setup of the new Centre, the company has just concluded a deal with one of the high street banks to get a loan of £30,000 on the 1st of August 2020. The interest on this loan will be 3.5% to be paid every month. The company will be required to make 12 equal payments to repay the loan starting end of September 2020.Financial informationAs mentioned above the company plans to sell a total of 1,200 units of the service packages between 1 August and October 2020. The fixed costs for the period are as below:Rent£ 18,000Cleaning£ 1,700Loan Interest£ 3,150General insurance£ 7,500Light and heating£ 4,200Local authority charges£ 4,550Fixed cost specific to each service packageBLFLMarketing20,500£ 25,000Admin costs£ 9,500£ 11,500Staff Salary£ 18,000£ 25,000From their costs estimates, the variable costs of the services are £150 for the BL and £200 for the PL. The fixed costs are for the whole period, so they are not affected by the level of service. However, the variable costs will increase with services output (ie sales output multiplied with variable cost per service package).Revenue from the sale of BL and PL will be on the basis of 40% cash in the same month, and the remaining 60% credit to be paid the following month.
Requirement:You will be required to write a management report to the management of Jackson Ltd directors, discussing the following issues: Provide an explanation on the different sources of funding the company can have and their advantages and disadvantages. You should make recommendations as to how the company can manage the same to help in the planned expansion program.Analyse the Investment proposals by using NPV and provide recommendations. You should also briefly comment on other investment proposal techniques that Jackson Limited may use, and the limitations of using those techniques 
The use of management tools such as Breakeven analysis and Budgets. 
A computation of your breakeven analysis and the cash budget for the first 3 months.An evaluation of the estimated company performance or position during the same period.A detailed Literature Review of the tools you have used such as breakeven analysis and 
budgets and their importance to business.Other issues for management to consider that you think are vital for them to survive and make a profit.Assignment GuidelinesStructureYou have been asked to produce a report. It should contain the following:Appropriate coversheet (as attached in this document)Title Page, including the given title in full.Executive Summary 
Contents Page 
Literature review to support your accounting models used. 
Sources of Funding 
Investment appraisal 
Cash budgeting 
Breakeven analysis 
Any other issues to be considered. 
Conclusions and Recommendations 
Appendices which should be numbered.Make sure you refer your reader to them as required.LayoutYour work should be word processed in accordance with the following:Font style, Arial, font size 121.5 line spacing.The page orientation should be ‘portrait’Margins on both sides of the page should be no less than 2.5 cmPages should be numberedYour name should not appear on the script.Additional SkillsAttend a Spotlight session as advertised here on Moodle.Marking Criteria/RubricPlease see the attached rubric with breakdown of the evaluative criteria that will be used for assessing this assignment.
CU Explanation on The Different Sources of Funding the Company Can Have and Their Advantages and Disadvantages Discuss

Effect of Mycotoxins on Animals. Mycotoxins are secondary metabolites produced by fungi. Secondary metabolites are chemicals produced by fungus that is not essential for growth of the animal. A mycotoxin is a fungal metabolite that causes an undesirable effect in exposed animals. While all mycotoxins originate from a fungus, not all toxic compounds produced by fungi are not characterized as mycotoxins. These unwanted effects are a result of the animal being in the state of mycotoxicoses, exposure often most likely from consumption. Damage can be caused to bodily systems vital for overall health function. The immunity of the animal is suscept to decrease rapidly under mycotoxicosis. Also, the reproduction will experience a decline in production due to the intake mycotoxins. Molds is fungi that develops into multicellular colonies. Molds are aerobic, which means they can only grow with the presence of oxygen. Molds can grow on silage, or wet feeds. Once molds begin to grow mycotoxins can be produced pre harvest, during storage, during transport, processing or feeding. Mold growth and mycotoxin production is related to inadequate storage methods, weather extremes, low quality of feedstuffs and faulty feeding conditions. Molds can grow in a pH range between four and eight. Also, molds can grow within a temperature range from fifty to up to one hundred and four degrees’ Fahrenheit. Mycotoxins can be detrimental in a variety ways depending on the type of mycotoxin becomes present. Mycotoxins are known to have variants that are carcinogens. A carcinogen is a substance capable of causing cancer in living tissues. Symptoms in animals can have a wide range of elements from decreased feed consumption, and even decreased metabolism of feed. Also weight loss is often seen in cases, weight loss, diarrhea, and vomiting. When breeding animals for their meat producers often see the problem of abortion and still births in animals who have consumed mycotoxins. Tissue necrosis and tumors are both symptoms that may arise within an animal who has consumed a certain type of mycotoxin. Tissue necrosis is known to be a characteristic or feature used when the highest grade of tumors is diagnosed. Tissue necrosis is a process of breakdown of ordered structure and function following traumatic damage, that is often irreversible. Necrosis occurs when nutritional demands of a growing tumor surpass the nutrient supply from the vasculature. Cell necrosis is recognized by changes in the nucleus that are microscopic. Changes can include the nucleus swelling, which will cause condensation of the nuclear chromatin. Mycotoxins effect on horses Mycotoxins effects on horses can be broken down into two generalized categories. Horses can either suffer from mycoses or even mycotoxicoses. Mycosis is when the growth of fungi becomes apparent on the animals’ body. Mycotoxicoses is more of when mycotoxins have been exposed to the horses diet or feed, respiratory system, or even dermal exposure can result in mycotoxicoses. Therefor mycotoxicosis deals more with once the mycotoxin has effectively entered a bodily system of the horse. Often symptoms of mycotoxicosis that will not be visible are reduced glycogen synthesis and impaired lipid transport. The most common for of mycoses in horses is known as guttural pouch mycosis. The fungus Aspergillus is the most common found fungus to be the catalyst for this fungal infection. The guttural pouch of the horse is one of a kind, this body part can only be found in horses and few other species. Horses in fact have two of these pouches and are extensions of the eustachian tube. Each guttural pouch is divided into two separate compartments. Plaques formed from the fungus form inside the guttural pouch, often along the internal carotid, external carotid, and maxillary arteries. Progressively the fungus has the potential to erode through the walls of these major blood vessels. Hemorrhage can result if proper care is not administered, however these hemorrhages may be fatal depending on the severity of the infection. If left untreated the infection will intensify until ultimately a fatal bleed occurs. There are many types of mycotoxins that have shown to impact horses in a negative way, and studies elude to this same information. Aflatoxin are known to be problematic to horses, these mycotoxins usually target the liver. Much like the aflatoxins, mycotoxin ochratoxin may be produced by aspergillus. Fumonisin is a mycotoxin produced by fusarium moniliforme that is known to have similar characteristics as most mycotoxins, and if improperly treated can result in fatality. Ergot is a mycotoxin produced by Neotyphodium, infection of this fungus can lead to an expected result of a decrease in performance and dystocia (difficulty giving birth). Ergot is oldest mycotoxin to date, this mycotoxin derives from the fungus Claviceps (Claviceps purpurea). This fungus is known to grow on the ears or rye and other cereal grasses. Ergot alkaloids are a unique sub set of secondary metabolites and have been identified by three groups as: ergopep times, clavines, and amides of lysergic acid Ergot is composed of alkaloids that may disrupt stimulation of smooth muscle cells. Upon consumption of this mycotoxin on feedstuffs horses will undergo ergotism. Ergotism is a term used for ergo-alkaline toxicity, these toxins are known to affect the endocrine, vascular, and nervous systems. The amount necessary to cause major impacts in horses is rather minimal. For symptoms to be triggered the build up of these mycotoxins only has to reach between fifty to one hundred parts per billion. Symptoms often vary from but are not limited to: abortion late in gestation, a decrease in lactation yields, and overall body function complications for foals. Ergot usually is a byproduct of malpractices of feed storage or even during harvesting. Fusarium is a mold that is been researched and is known to grow on corn, wheat, and grasses. Deoxynivalenol or DON is the mycotoxin that is most likely to be produced from the mold Fusarium. Deoxynivalenol is a vomitoxin, therefore once this mycotoxin has either been inhaled or consumed by the horse multiple unwanted symptoms will arise. This mycotoxin upon entering the body is associated with acute diarrhea, ataxia, vomiting, and if untreated can lead to a sudden death. However due to the monogastric intestines of the animal horses have a high tolerance for deoxynivalenol and are usually less suscept. Yet high amounts of deoxynivalenol when consumed in higher quantities horses are known to reduce feed intake. Generally once feed intake has decreased work or performance declines just as well. The pivotal aspect of mycotoxins comes about when the possibility of different mycotoxins originating from the same fungus. Fusarium moniliforme for example produces the mycotoxins known as FB1, FB2, and FB3. All three of these mycotoxins in particular are prone to become present in corn crops. Within in horses FB1 is one of the more devastating mycotoxins because of the known relations between consumption of this mycotoxin and its relevance to the development of the neurological disease leukoencephalomalacia. The danger of leukoencephalomalacia lies within its ability of transmission to an entire herd.The symptoms of this disease can range from ataxia, neurological dysfunction, and even lethargy. Leukoencephalomalacia is such a detrimental disease due to there being no treatment or cure. Therefore producers of these animals should be weary of the risk factors associated with administering even a small amount to the animals’ diet. Mycotoxins effect on cattle Most mycotoxins are excreted by cattle by milk, feces, and urine. Mycotoxins in general are also adept to be chemically stable and can survive most food processing. The major mycotoxins that have shown to trouble cattle are: aflatoxins, ochratoxins, fumonisins, trichothecenes and zearalenone. The molds that these mycotoxins derive from are Aspergillus, Fusarium and Penicillium, these all can produce detrimental mycotoxins. Upon observation of an reproduction decline throughout the entire herd, mycotoxins may often be the primary agent. Deoxynivalenol or vomitoxin are produced by Fusarium graminearum. These mycotoxins will come about more frequently in a wet and cold environment. Vomitoxin in particular has shown over time to be most prevalent in wheat crops. Mycotoxins’ have shown to disrupt protein synthesis, and cell proliferation, and in extreme or untreated cases cell death is prevalent occurrence. Aflatoxin is another mycotoxin that is produced by the fungus Aspergillus flavus and Aspergillus parasiticus. This mycotoxin does have negative effects in cattle but, even humas as well may experience symptoms upon consumption. Aflatoxins have been well documented to be found primarily on corn crops. Once a bull or heifer have consumed corn with the mycotoxin aflatoxin present the animal will experience cellular changes, cell deregulation, and if left untreated the mycotoxin will run its course and cause fatality. Fumonisins are one cause of mycotoxins that is produced by various naturally occuring fungi. Fumonisins have a higher chance of reproduction in an hot, dry climate once the corn has started to sprout. However a wet and cool climate may lead to the growth of these toxins during the pollination stage of growth. Consumption of these mycotoxins in cattle is known to be associated with immunosuppression, oxidative stress, and leaky gut syndrome. Leaky gut syndrome is an ideology of loose connections within the cattles’ gut. The intestine of the animal is compared to that of tears within a coffee filter, which allows the release of important nutrients needed for absorption and metabolism. The important nutrients that are affected by leaky gut syndromes are the digestible proteins. However the body has an adverse response to this imbalance and immune cells attack the autoimmune invaders, which will often result in inflammation that can be painful to the animal. Ruminant animals are generally considered to be less suspect to mycotoxicoses than monogastric animals. The theory of how ruminant animals can avoid in feed mycotoxins is through the power of the rumen. The rumen flora which is composed of bacteria, fungi, and protozoa is what inactivates mycotoxins that may enter the animals’ system. The process in which ruminants break down ingested feed is through microbial fermentation. The most prevalent protozoans found in the rumen originate from the genuses of: Epindinium, Entodinium, Diplodinium, and Holotrich ciliates. On average between two hundred and five hundred thousand protozoa can be found in the rumen of these animals. Bacteria compose about half of the living organism that cohabitate inside the rumen. Anywhere between twenty to fifty million bacteria are likely to be found in the rumen of cattle. Even with the majority percentage of the living organism present in rumen are bacteria, bacteria do more than half the work that is required for microbial fermentation. Rumen bacteria are identified four distinct classifications. Bacteria in the rumen can be identified as either fiber digesters, starch and sugar digesters, hydrogen using bacteria, and lactate using bacteria. Bacteria and Protozoa digest roughly between seventy and eighty percent of digestible matter within the rumen. Mycotoxins effect on poultry Mycotoxins are fungal metabolite which upon ingestion, inhalation, or absorption through the skin of the chicken can cause disease to the animal and possibly can become fatal. Mycotoxins are toxic compounds that are naturally produced by certain types of molds. Young chickens are more sensitive to the presence of mycotoxins in there intestinal, or respiratory tract. There is a wide range of effects that have been studied in chickens, however immunosuppression. Immunosuppression is when the strength of an animals’ immune system is lowered, which in fact makes the animal more susceptible to infection or illnesses. There is a restricted level of mycotoxins allowable in animals within the United States. However even low levels of mycotoxins present in feed over long periods of time can impair the immune system, leading to immunocompromised animals. Ochratoxins are a gathering of fundamentally related metabolites that are derived by growths having a place within the general Aspergillus and Penicillium, and Ochratoxin (OTA) is the most pervasive mycotoxin of this gathering. Indications of OTA lethality in poultry incorporate shortcoming, weakness, diminished feed utilization, decreased development rate and egg generation, poor feathering, and extreme mortality at high dietary focuses (Hamilton et al., 1982; Gibson et al., 1989; Huff et al., 1988b). Pathophysiological changes incorporate diminished pee focus and glomerular filtration rate, debilitation of proximal rounded capacity, and degeneration and ultrastructural adjustments in renal uprightness (Huff and Hamilton, 1975; Glahn et al., 1988, 1989). Increments in the general loads of liver, spleen, pancreas, proventriculus, gizzard, and testicles have additionally been accounted for in poultry encouraged OTA (Gibson et al., 1989; Huff et al., 1988b). Ochratoxin A comprises of an isocoumarin moiety connected through the 7-carboxy gathering to the amino corrosive L-β-phenylalanine. At a cell level, OTA meddles with DNA, RNA, and protein combination by repressing the catalyst phenylalanine-tRNA synthetase (Marquardt and Frohlich, 1992). Ochratoxin An additionally influences renal starch digestion through a decrease of the renal mRNA coding for phosphoenolpyruvate carboxykinase, a key catalyst in gluconeogenesis (Leeson et al., 1995). The impacts of OTA on DNA, RNA, and protein combination are believed to be because of the phenylalanine moiety of the poison rivaling phenylalanine in the chemical catalyzed response (Marquardt and Frohlich, 1992). Ochratoxin additionally causes hypercarotenemia in grills (Huff and Hamilton, 1975) that is more extreme than that brought about by Aflatoxins. (Osborne et al., 1982; Schaeffer et al., 1987). Mycotoxins effect on swine Swine in particular are extremely sensitive to mycotoxin exposure. Young pigs and breeding sows and boars are more suscep to mycotoxins than more mature swine would be. However both the phase of life the animal is important, along with the phase of production the animal is in, and the level of mycotoxin concentration present in the feed. Likewise as there are many varieties of mycotoxins, there is within that a variety of mycotoxins that can potentially cause significant health and performance problems. These mycotoxins include: Aflatoxin, Ochratoxin, Deoxynivalenol, Zearalenone, Fumonisin. Furthermore, the fungi Aspergillus produces the mycotoxin most detrimental to pigs, which is claviceps. Mycotoxins can effect swine by either way of mycosis or mycotoxicoses. Mycosis is defined as any disease that is triggered by the development of fungi on the swines’ tissue. The fungi invades the tissue of the animal, which can result in disease. Mycotoxicoses is another way mycotoxins can negatively effect swine however, through a different route of entry. Mycotoxin exposure within digestive or respiratory system, even physical contact with the secondary metabolites is enough to inhibit an immune response. Prevention of mycotoxins Mycotoxins as expressed throughout the piece of work are harmful to animals and have negative effects of many sorts. Preharvest treatment is important and can be completed with the use of Afla-Guard and Aspergillus flavus AF36, show promise of reducing Aflatoxins in grain through competitive exclusion. Post harvest it best to lower or limit moisture content of plant seeds, after harvesting and during storage. Commodities should be stored at lower temperatures as much as possible. Pest control in stored bulk grains with approved insecticides will limit insect infestations. Also, the growth of fungi can be ceased by re drying the products, which are mycotoxins. If the fungi has abundantly covered feedstuffs is not uncommon to destroy the contaminated products by way of burning. With the inclusion of additives such as sodium bentonite that serve as binders. Efficient irrigation can reduce mycotoxins formations in the field before the crop is harvested. Organic acids can be used as preservatives for high moisture feeds. Conclusion Mycotoxins are secondary metabolites created by fung. While all mycotoxins begin from an fungi, not every poisonous compound are portrayed as mycotoxins. These undesirable impacts are a consequence of the creature being in the condition of mycotoxicoses, presentation frequently no doubt from utilization. Likewise, the propagation will encounter a decrease underway because of the admission mycotoxins. Molds form into multicellular provinces. Molds are vigorous, which implies they can just develop with the nearness of oxygen. Molds can develop on silage, or wet feeds. When molds start to develop mycotoxins can be delivered pre collect, amid capacity, amid transport, handling or bolstering. Shape development and mycotoxin generation is identified with insufficient capacity strategies, climate boundaries, low nature of feedstuffs and broken encouraging conditions. Molds can develop in a pH extend somewhere in the range of four and eight. Additionally, molds can develop inside a temperature go from fifty to up to one hundred and four degrees’ Fahrenheit. Mycotoxins can be inconvenient in an assortment ways relying upon the sort of mycotoxin winds up present. Mycotoxins are known to have variations that are cancer-causing agents. A cancer-causing agent is a substance fit for causing malignancy in living tissues. Side effects in creatures can have a wide scope of components from diminished feed utilization, and even diminished digestion of feed. Additionally weight reduction is frequently found in cases, weight reduction, loose bowels, and retching. When rearing creatures for their meat makers frequently observe the issue of premature birth and still births in creatures who have devoured mycotoxins. Work Cited Foulk, Donna. “Mold and Mycotoxins in Horse Hay.” Penn State Extension, 23 Mar. 2019, Wright, Dr. Bob. “Molds, Mycotoxins and Their Effect on Horses.” Fungal Mycotoxins of Grain and Their Potential Effect on Horses, 2003, Bennett, J W, and M Klich. “Mycotoxins.” Clinical Microbiology Reviews, American Society for Microbiology, July 2003, Logue, Jeff. “Equine Leukoencephalomalacia.” Equine Leukoencephalomalacia, 1997, TRICHOTHECENE MYCOTOXIN, “Trichothecene.” Trichothecene – an Overview | ScienceDirect Topics, Effect of Mycotoxins on Animals
NURS 6630 WU Wk 7 Elderly Widow Who Just Lost Her Spouse Case Discussion.

I’m working on a nursing question and need a sample draft to help me understand better.

Please respone Mariane Sule RE: Discussion – Week 7 Initial postCOLLAPSECase: An elderly widow who just lost her spouse. Three questions to ask the patient and a rationale for asking these questions.The three questions I will ask the patient are: 1) Have you ever had any past history of depression. 2) How do you cope day by day after the loss of yours? 3) Do you regularly take your medication? The reasons for asking those questions are because: It’s important to know what the patient’s mental health has been like before her husband’s death or if the patient may be experiencing recurrent depression. It’s equally important to know if the patient has any support system after her husband. Because the lonely she becomes the more difficult it is for the patient to find comfort or have better coping mechanisms. The reason for asking if the patient regularly takes her medication is to find out how much the patient is affected by the actual situation of “mourning her husband”. Because people who experience difficult situations such as mourning, depending on the relationship they have, the window may end up losing interest in everything including taking the daily medication. It is also important to know if he is physically incapacitatedPeople in the patient’s life to speak to or get feedback from to further assess the patientI would elicit this information first and also find out if she has any children and or possibly a guardian. Depending on this information and whom; I would want more information and clarification on her living situation, socioeconomic status. I would ask about a support network- does she have friends/family, someone to talk to. I would ask her about her sleep habits, does she nap during the day? What does she do before going to bed? Physical and diagnostic testsThe type of physical exam that would be appropriate for the patient would include a heart rhythm check, an assessment of the eye, and her oxyhemoglobin saturation. Diagnostic checks suitable for the patient would include actigraphy tests and polysomnography tests. The main aim of these tests will be to measure the sleeping patterns of the patient. polysomnography test looks into the patient’s sleep pattern while actigraphy measures total sleep time, sleep latency, and sleep patterns.Differential diagnoses for the patientThe patient’s differential diagnoses include generalized anxiety disorder, major depressive disorder (MDD), and restless leg syndrome. MDD would likely explain why the patient is currently experiencing insomnia. Some key takeaways would be difficulty in concentration, loss of appetite, and hopelessness (Keller et al., 2019). If these symptoms would be available, then she would be diagnosed with insomnia. After her husband’s death, the patient might have developed depression and later affected her sleeping pattern. Patients that are experiencing sleeping disorders are likely to be diagnosed with sleeping disorders (Bollu & Kaur, 2019).Pharmacologic agents and their dosingSelegiline The transdermal patch would be administered with the patient being advised to take a 6mg daily dose applied every day. If she doesn’t improve, then an increase of dosage by 3 mg daily for two weeks will be prescribed till the total dosage reaches 12 mg daily. The purpose of Selegiline The transdermal patch will be to inhibit the monoamine Oxidase-B isoenzyme (Puttrevu et al., 2020).Eszopiclone with a dosage of 3 mg administered orally before the patient goes to bed is prescribed. The drug is recommended for the fight against insomnia as it reacts with the gamma-aminobutyric acid (GABA) receptors at the allosterically binding sites at the benzodiazepine receptors. The drug method of action is to slow down brain activities hence allowing sleep i.e., induce sedation and hypnosis to allow sleep (Rösner et al., 2018).Drug therapy contraindicationsOne of the contradictions of using Selegiline The transdermal patch would be the increased risk of serotonin syndrome i.e., taking selective serotonin reuptake inhibitors, mirtazapine, and other vasoconstrictors and analgesics (Puttrevu et al., 2020).CheckpointsThe patient will be assessed after a four-week interval to determine if the symptoms have improved. If the symptoms have not improved after four weeks, selegiline transdermal will be increased by 3mg for four weeks until 12mg which is termed as the maximum dosage. If within the first four weeks the symptoms would have changed positively, then the dosage is maintained till the patient completes the dosage in which the sleep problem would be effectively managed. ReferencesBollu, P. C., & Kaur, H. (2019). Sleep Medicine: Insomnia and Sleep. Missouri medicine, 116(1), 68–75.Keller, A. S., Leikauf, J. E., Holt-Gosselin, B., Staveland, B. R., & Williams, L. M. (2019). Paying attention to attention in depression. Translational psychiatry, 9(1), 279., S. K., Arora, S., Polak, S., & Patel, N. K. (2020). Physiologically Based Pharmacokinetic Modeling of Transdermal Selegiline and Its Metabolites for the Evaluation of Disposition Differences between Healthy and Special Populations. Pharmaceutics, 12(10), 942.ösner, S., Englbrecht, C., Wehrle, R., Hajak, G., & Soyka, M. (2018). Eszopiclone for insomnia. The Cochrane database of systematic reviews, 10(10), CD010703. QUOTE EMAIL AUTHORSelect: All None
NURS 6630 WU Wk 7 Elderly Widow Who Just Lost Her Spouse Case Discussion

Read the scenario and then answer the questions. You need to respond to another juror(classmate) to get full credit.

Read the scenario and then answer the questions. You need to respond to another juror(classmate) to get full credit. Remember to cite the text at least once to support your decision. As usual, spelling and grammar count. The case of the crazy Lawnmower Paul, a mechanical engineer, bought a lawnmower from Lawncore Co. for $499. The salesman said it was an excellent quality lawnmower and even professionals used the same one. There had never been a complaint. It had a 30 day limited warranty on parts and labor. There was also a disclaimer that the manufacturer, designer and retailer were not responsible for any injuries caused by improper assembly or use. Everything was assembled by Lawncore, except the handle needed to be attached at home. The mower was assembled on March 1. Paul did not get a chance to use it until April 4 because he had to go out of state due to the death of his brother. On April 1 when he started cutting his lawn, a blade broke off and cut his neighbor who was looking at the new lawnmower. Then the lawnmower suddenly stopped working. The neighbor’s leg was cut. Paul did not get to finish cutting his lawn. He took his neighbor to the hospital. The neighbor’s medical bills amounted to $10,000. Paul took the lawnmower back to Lawncore Co. to tell it of the injuries to his neighbor and to get it repaired. The salesman first said it was too late as the warranty was only for 30 days. Paul said , but “it does not work” and it caused an injury to the neighbor. The salesman then said that it was Paul’s fault because he must have improperly assembled the handle. You are on the jury. You need to answer the two questions below. Who is responsible for the injuries to the neighbor? Explain. Have any warranty(s) been breached? Explain which ones, how and why?

A Critical Analysis of Presidential Campaign Advertisements

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A Critical Analysis of Presidential Campaign Advertisements
Introduction. The 2012 presidential campaign broke all records for campaign advertisements during an election campaign. The total amount spent on television advertising on the two major party candidates reached $900 million with over 1 million ads being aired. (Washington Post’s Analysis on Campaign Ad Spending) The vast majority of this money is being spent on ads that are commonly no more than 30 seconds long, and yet they are often credited with tremendous influence in determining the winning candidate. In the half-century since presidential candidates first started using television advertisements, these “spots” have become a vital part of any political campaign.
Background. Campaign ads are products of the time periods in which they were made. It can be said that they are historical artifacts – whether they address timely issues or more general themes, they can help teach us what mattered to voters in a given campaign. And when we look at a sampling of campaign advertisements from the past 60 years of presidential campaigns, we can come to an understanding of the extent to which presidential elections, the media and the issues that matter to voters has changed.
Your task: Today we are going to take a close look at campaign advertisements from several elections over the course of the second half of the twentieth century. Choose an election year to learn about

Use the website from the Museum of the Moving Image called The Living Room Candidate (
Read the summary of the election in the box on the right side of the screen – this will provide you with the historical context necessary to understand the advertisements.
Browse through the campaign advertisements from the major party candidates in your Chosen election year.
Choose ONE advertisement to present to the class as being the most effective AND explain why you considered it to be effective. Use the worksheet below to help form your reasoning.
Be ready to present your findings to the class.

A Critical Analysis of Presidential Campaign Advertisements

Problems of the Poorest People, Capital Flight, and Migration Essay

Introduction Globalisation is probably on of the most controversial issues in the world history: many people find it as an amazing possibility to achieve success and make use of a number of opportunities, still, some people, for example the citizens of South Africa, suffer a lot because of inabilities to solve their problems and eliminate poverty that decreases the level of human lives considerably. There are many writers and researchers like Dicken (2007) or Ravenhill (2008) who investigate the economic perspective of globalisation, however, not all of them manage to discuss the chosen issue in a proper way. In this paper, the work by a famous British professor, Paul Collier, will be evaluated in order to prove that the poorest billion of people have already integrated into the world economy by means of capital flight and migration, still, the outcomes of such integration are not as successful as they are in the countries like the USA or China. The Bottom Billion: Why the Poorest Countries Are Falling and What Can Be Done about It is the book where the author evaluates the level of developmental aid offered to the developing countries like Africa and underline the advantages and disadvantages of the process of globalisation. Africa has bad governance, one of the four traps defined by Collier in the book, that is not ready to use the full potential of the country. This is why the citizens of South Africa have to make use of capital flight and migration in order to continue developing and becoming a recognisable country on the world’s arena. Collier’s attempt to discuss the difficulties existed in developing countries is powerful indeed; poverty is the challenge that has to be overcome by the Africans by means of proper understanding of the main ideas of globalisation, four traps which are dangerous for the country, and the possible instruments like capital flight or migration, which may be used to overcome poverty and find another way to success. Poverty as the problem to be solved Poverty is the challenge that not all nations are able to overcome. Sachs (2005, p. 10) admits that even if some countries like China or Canada are lucky enough to overcome poverty and stabilize citizens’ lives, it does not mean that poverty will never influence human lives again or create some new risks for human survival. Get your 100% original paper on any topic done in as little as 3 hours Learn More It is wrong to believe that poverty may be completely illuminated from human lives, and in order to succeed in development, people have to understand the main reasons of poverty and the conditions under this problem may appear again. However, there are a number of countries where poverty is not that huge problem anymore; China, Canada, the USA are the best examples of such countries. Collier explains that even such developing countries like Africa are able to create a certain paradigm by means of which people can shift away poverty and set new goals to be developed. Acemoglu and Robinson (2006) discover that “the European presence in South Africa began in 1652 when the Dutch East Indian company founded a colony in Table Bay” (p. 10). It was a kind of the starting point after which the country was not able to control the activities which took place on its land, and the Africans could help but fighting poor conditions of living. Types of traps for people In order to understand how poorest people may integrate in the world economy through capital flight and migration, Collier offers to define poverty as not the only trap which the countries may fall into. What he suggests is to pay more attention to the four types of traps which are dangerous for different countries. These traps are the conflict trap, the natural resource trap, the bad governance trap, and the landlocked with bad neighbours trap (Collier 2007, p. 5). The author underlines that poverty cannot be intrinsically a trap as much more countries remain to be poor; this is why Lesotho, Botswana, and other countries in the South Africa still get a chance to overcome this challenge in case they clearly identify what else may lead to poverty. The citizens of South Africa are actually in several traps at the same time, still, the most terrible trap for the country is the one of bad governance. In a number of small countries, governance plays a very important role in promoting economic development. However, it happens that small governments are usually eager to achieve some personal benefits and decide to corrupt or choose some bad policies. This is why it is hard to understand how economic development may occur under such conditions. It is necessary to admit that South Africa as well as the rest of the countries on the continent is characterized by impressive workforce that may be developed along with the coast. Unfortunately, the government of the country does find it beneficial to make use of natural sources but rely on aid-flows which may be received from different developed countries. We will write a custom Essay on Problems of the Poorest People, Capital Flight, and Migration specifically for you! Get your first paper with 15% OFF Learn More The bad governance trap is not the only one that exposes to danger the whole country. Collier says that the landlocked with bad neighbours trap may also challenge the country. Most African countries have to be geared to the world market or be enclosed. And even if some countries have appropriate geographic location, its citizens are not able to benefit from this natural gift. For example, African economies are more inward looking, this is why when the time to increase its neighbour comes, the vast majority of Central African countries which are far from the coast do not have enough sources to take the step required and the countries suffer because of such landlocked trap. Switzerland is another example of a landlocked country; still, it continues advantaging from cooperating with Germany and Italy in the market sphere. Though the natural resource trap is not as dangerous for Africa as for other billion countries, the government cannot find practical application to the already defined sources. This is why even this trap may become dangerous for the country under consideration. The last trap that plays a crucial role in economic development in Africa is connected to the conflicts which are inherent to the country. Brett (2008) mentions that during the 1970s, post-colonial Africa overcame the economic crisis that was based on the “intense conflict over political control and resource allocation, and a shift to military or authoritarian rule” (p. 340). The possibility to escape the traps The peculiar feature of the book introduced by Collier is that the author does not lose hope to improve the economic conditions in billion poorest countries and does offer a number of ideas of how and when the country should start its development. The examples of how African countries may be developed are observed in the successful development of Botswana that “had the highest rate of per-capita growth of any country in the world in the last 35 years” (Acemoglu, Johnson,

Writer’s Choice

Writer’s Choice. Paper details This assignment must be completed on the template provided. This assessment requires you to explore the literature to find 2 research articles that are examples of 2 different levels on the levels of evidence hierarchy. The pyramid that illustrates the levels of hierarchy is on page 13 in your textbook. Please read about the levels of evidence to ensure you understand them before selecting your articles. Objectives Understand the research levels of evidence hierarchy (pyramid) Demonstrate effective data base search strategies Identify studies by level of evidenceWriter’s Choice

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