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Educate People On Pusher Syndrome

The brain is a very complex organ. Everything we do or think takes place in a very confined, small space. Any injury that occurs in the brain can affect the whole body. Pusher syndrome is a disorder following right or left brain damage. Pusher syndrome occurs when the patient pushes away from the nonhemiparetic side.2 Pusher syndrome is observed in about 10 percent of acute stroke patients that has hemiparesis.10 Typically, pusher syndrome occurs in strokes, but may also occur in trauma, tumors, or other kinds of brain damage.11 Pusher syndrome usually occurs when the posterolateral portion of the thalamus, that is located deep within the cerebral hemispheres beneath the cortex and is the relay center for sensory and motor mechanisms and the suprathalamic white matter is involved.1,12,13 The damage is caused by higher pressure, swelling, and other secondary pathologies.10 Pusher syndrome is usually caused by a hemorrhagic stroke rather than a cerebral infarction.10 A posterior thalamic hemorrhage causes bigger lesions than thalamic infarctions, which frequently leads to pusher syndrome.7 Stroke Since pusher syndrome is usually the result of a stroke, it is important to know some basic facts about strokes. One of the most common causes of death in the United States is due to a stroke.14 The long term cause of disability is also due to a stroke.15 Strokes can cause an increased dependence for many survivors.16 A stroke is one of the most expensive and life changing syndromes keeping people from fully participating in their lives.17 Stroke occurs when there is an interruption of blood flow to the brain and it causes sudden severe neurologic loss of function. Hypertension, heart disease, and diabetes are major risk factors for having a stroke.5 The age of people having strokes are starting to decrease.17 There are two different classifications of stroke, which are hemorrhage and ischemic strokes. Ischemic strokes are caused by thrombosis, embolism, or systemic hypoperfusion, while hemorrhage strokes are caused by intracerebral or subarachnoid hemorrhage. A thrombotic stroke is caused when a disease causes a thrombus and reduces blood flow distally. An embolic stroke is caused when debris breaks off and travels elsewhere to block arterial access to a particular region of the brain. Intracerebral hemorrhage stroke is usually from small arteries bleeding directly into the brain. This bleeding causes a localized hematoma that spreads along white matter pathways. The accumulation of blood can occur within minutes or hours. The hematoma grows until the pressure increases to its limit or until the hemorrhage decompresses by emptying into the ventricular system or into the cerebrospinal fluid on the surface of the brain. Subarachnoid hemorrhage stroke is usually caused by a rupture of arterial aneurysms that are located at the base of the brain. Subarachnoid hemorrhage strokes can be also caused by bleeding from vascular formations that lie near the pial surface of the brain. Ischemic cerebral infarctions are responsible for 80 percent of strokes and brain hemorrhage is responsible for 20 percent of strokes.18 Strokes can cause many neurological deficits. Those deficits include deficits in motor control, abnormal synergistic patterns of movement, muscle weakness, sensory deficits, and a loss of range of motion.14 People having symptoms of a stroke should have a MRI or CT scan within 24 hours of onset.19 Symptoms of stroke include: sudden numbness or weakness in the arm, leg, or face on one side of body, sudden confusion or trouble speaking, sudden trouble seeing, sudden difficulty with walking, dizziness, loss of balance, and sudden headaches.5 Characteristics of Pusher Syndrome Pusher syndrome is characterized by pushing toward the hemiparetic side. A patient with pusher syndrome strongly resists the vertical upright position. They align their longitudinal axis of their bodies with what they perceive as vertical, which is toward their hemiparetic side.3 A patient with pusher syndrome usually tilts their body 20 degrees to their hemiparetic side.4 Pushing varies in severity and increases with postural challenges. In sitting, the patient leans toward the weaker side. In standing, the patient has a high risk for falls because they are unbalanced and the hemiparetic lower extremity cannot support the weight of their body. The patient shows no fear of pushing to the weak side.5 These patients actually show fear of falling toward their nonparetic side and that is why they push toward the hemiplegic side.20 Normally, a patient with a stroke increases their weight bearing on their stronger side, so this syndrome is opposite of the expected tendency’s.5 Pusher syndrome is more prominent when patients are upright rather than lying down.21 Patients with pusher syndrome has paresis of the contralesional extremities more frequently and more severe than patients without pusher syndrome13 These patients also have an unstable gait because they continuously fall to their paretic side. The inability to bear weight on the paretic lower extremity also causes gait disturbances. Part of the underlining mechanism of pusher syndrome is the mismatch between the visual vertical and tilted orientation of the body.14 Diagnosing Pusher Syndrome To diagnose pusher syndrome, the Standardized Scale of Contraversive Pushing (SCP) is used on the same day of the MRI acquisition. The SCP analyzes three different areas. The first area to be assessed is symmetry of spontaneous body posture. The next is the use of the nonparetic arm or leg to increase pushing force by abduction and extension of extremities. Last is the resistance to passive correction of posture. These tests are determined when the patient is sitting with feet on the ground and standing. For a patient to be diagnosed with pusher syndrome, all three analyzed areas have to be present and with a score of at least one with respect to their spontaneous postures and at least a score of one with respect to the use of the nonparetic arm and leg to increase pushing force by abduction and extension. Also the patient has to show a resistance to the correction of the posture.6 The SCP is a simple and fast test but it is not suitable when symptoms are slight and only show up in dynamic activities like walking. Another way to diagnose pusher syndrome is a four-point scale that assesses the presence of pusher syndrome by examining different postures. If a patient does not have pusher syndrome they will receive a score of zero. If pusher syndrome is only present in standing, the patient receives a score of one. If pusher syndrome is also present in sitting the patient receives a score of two. If pusher syndrome is also present while lying down the patient receives a score of three.7 Treatment of Pusher Syndrome Physical therapy is a very important part of recovery for a patient with pusher syndrome. Patients with hemiplegia and pusher syndrome will be admitted to inpatient rehabilitation more frequently than patients with less severe symptoms.7 Pusher syndrome causes impairments on postural balance.8 One of the first goals of physical therapy should be to demonstrate and align posture.2 Visual cues may be helpful for patients to try to align their body axis to the earth vertical.14 The therapist can sit next to their less involved side or have the patient sit against a wall with their less involved side and tell the patient to lean toward the therapist or against the wall. To help with sitting posture, physical therapy can include sitting on a therapy ball to promote symmetry and sitting. While the patient is on the ball, the weaker lower extremity should cross over the stronger lower extremity. To help get the weak lower extremity out of flexion, which is often the position of the weaker lower extremity; the patient can wear an air splint or a leg splint. The therapists can actually tap directly over the quadriceps muscle to promote extension. A modified plantigrade position is a great position to begin early standing. In this position, the therapists can focus on using the weaker lower extremity to work on unilateral support. The weaker upper extremity may also be in a position of flexion, so an air splint can be used to promote extension of that upper extremity. A patient can stand in a corner or doorway to promote symmetrical standing. The therapist should block the stronger extremities from moving into abduction and extension and pushing. The therapist should provide constant feedback about body orientation and have the patient practice correcting orientation and weight shifting.5 When a patient begins gait training, the therapist can lower the height of the assistive device so the patient has to bear weight on the uninvolved side.22 If a patient requires transferring, they should be transferred to their weaker side. Transferring this way is much more convenient since the patient is already pushing in that direction. Also, since pusher syndrome is the result of a stroke, the treatment of a patient with a stroke should also be discussed. The level of the patient with a stroke must be part of consideration when a patient begins therapy. There are many tests to measure the independence in activities of daily living. These tests include the Functional Independence Measurement (FIM), Glasgow Outcome Scale (GOS), modified Rankin Scale (mRS), and the Barthel Index (BI).23 A therapist will examine the patient and make a decision on the level that the patient is on in order to see which direction to begin therapy. Another scale that needs to be evaluated for a patient with a stroke is the Brunnstrom stages of recovery. This scale rates the patient in the progression of the typical characteristics of stroke behaviors. This scale goes through seven stages of recovery. The stages begin with stage one as the patient is flaccid, stage two as the patient begins to develop spasticity, stage three as spasticity is at its greatest, stage four and five as the spasticity decreases, stage six as spasticity is completely gone and stage seven as the patient is back to normal function. The therapist should also be aware of synergy patterns and help the patient to work out of these patterns.22 The stages of motor control and the stages of developmental posture are very important aspects of therapy for stroke patients. A therapist should be aware of these sequences and follow them in the treatment of a stroke patient.5 After a patient suffers a stroke, balance ability can be improved by physical therapy interventions. After a stroke, early impaired balance is strongly associated with future function and recovery.15 The Bobath concept of Neuromuscular Developmental Treatment (NDT) is one of the many tools that therapist can use to deal with individual deficits and opportunities for stroke survivors. NDT is especially useful for those patients with a good prognosis for recovery. Bobath explained that a patient suffering from hemiplegia should be active while the therapist assists them in moving by using key points of control and reflex inhibiting reflexes.9 The key points of control are head, shoulders, hips, or distal extremities. The shoulder and pelvic girdle is the most important points to influence postural alignment. A therapist would apply manual contact to the shoulder and pelvis to influence muscle tone distribution and distal movements. The distal key points are the elbows, hands, knees, and feet. The distal key points affect the movement of the trunk. Once a patients’ tone is manageable, the therapist superimposes normal movements and posture. When a therapist superimposes normal movement and posture, it is done within the context of a functional activity. NDT is a great way to inhibit abnormal postural reflex activity and movements and facilitate normal patterns. Normal motor patterns include head and trunk control, upper extremity support, and balance reaction. NDT is also a good approach to align posture.22 Proprioceptive Neuromuscular Facilitation (PNF) is reported as being the most effective protocol for achieving the greatest increase in range of motion.24 PNF is also used to increase strength, flexibility, and range of motion. By increasing these things and integrating these gains, the patient can establish head and trunk control, iniate and maintain movements, control shifts in the center of gravity, and control their pelvis and trunk while the extremities move. PNF is unique diagonal patterns of movement. Most movements do not occur only in the cardinal plane but also occur as triplanar. PNF patterns simulate the demands incurred during functional movements.22 Another good approach to physical therapy is to strengthen the weak muscles. Tone is another issue that must be addressed in physical therapy in a patient with a stroke and pusher syndrome. Patients can either have low tone (flaccid) or high tone (spastic). Low tone can be corrected by using facilitory techniques, and high tone can be corrected by using inhibitory techniques.5 Some facilitory techniques include quick stretching, tapping, vibration, approximation, and weight bearing. Some inhibitory techniques include slow, rhythmic rotation, weight bearing, prolonged icing, and static stretch.22 Occupational and Speech Therapy Occupational therapy is also required to expand rehabilitation to address participation in work, family, and community life.17 Upper extremity weakness also needs to be strengthened by the occupational therapist to perform activities of daily living.25 Although speech therapy may not be needed to treat pusher syndrome patients specifically, the injuries that result in this condition will require speech therapy, such as stroke or brain injury. Speech therapy may be needed to address aphasia, global or expressive. Prognosis of Pusher Syndrome Even though progress of a patient with pusher syndrome is based on a patient by patient case, they usually have good results. With effective training, the potential for minimizing the impact of pusher syndrome is good. Motor learning strategies are also very effective in reducing the effects of pusher syndrome and enhancing recovery.5 The functional recovery process may be very slow and require a longer stay in the hospital but usually a patient with pusher syndrome makes a full recovery. Recovery is usually completed by six months after the stroke.7 Conclusion Pusher syndrome can be a very devastating symptom after a hemorrhagic stroke. Ten percent of acute stroke patients suffer from pusher syndrome. A patient with pusher syndrome can have greater challenges with function and mobility. They have difficulties with standing and sitting as they push to their hemiparetic side and resist correction of posture. The Standardized Scale of Contraversive pushing (SCP) and a four-point scale are two ways to diagnose a patient with pusher syndrome. The treatment of pusher syndrome is dependent on physical therapy. Initially, correcting posture is the main focus of therapy. Then balance, strengthening weak muscles, and correcting abnormal movements are the focus of therapy. Neuromuscular Developmental Treatment (NDT) and proprioceptive Neuromuscular Facilitation (PNF) are great tools that help with physical therapy. Patients with pusher syndrome may have a slower recovery and a longer hospital stay, but usually make a full recovery within six months.
South University Actions Of The Florida State Sheriff Memorandum.

I’m working on a law case study and need an explanation to help me understand better.

Assignment: Negligence Case — AnalysisFamiliarity with the elements of negligence is crucial to the personal injury paralegal’s knowledge base. Also important to any paralegal’s tool box of skills are legal research and writing. The following scenario will help you exercise those skills by applying what you have learned in this week’s lectures and materials.A state statute allows handgun owners to carry concealed handguns on the condition that the handgun owners obtain a permit from the county sheriff for the county in which they reside. The sheriff is prohibited from issuing a permit for a period of three days after receiving the application. Sheriffs cannot issue a permit to a minor, a felon, or a person who is or has undergone treatment for any psychiatric condition.Alan Allen, aged 32, applies to the sheriff for a permit. He and the sheriff are friends, so the sheriff issues the permit without waiting three days or asking Allen if he has or is undergoing treatment for any psychiatric condition. In fact, Allen has been treated for severe depression and currently is taking medication for the same. Allen goes to a local tavern for a drink, gets into a fight with Chuck Charles, and pulls his gun, intending to frighten him. The gun fires, hits and shatters a bottle behind the bar, sending shards of glass flying. A piece of glass lodges in Chuck Charles’ eye, blinding him in one eye.Draft an internal memorandum on the case to your attorney using Lexis Advance to find primary sources of law, such as cases and statutes on point. You can use the law of your state, even if it differs somewhat from the fictitious state statute mentioned in the fact pattern. Cite all sources using the Bluebook format. Address in the memo the following questions:Are the sheriff’s actions negligent?If so, is that negligence the proximate cause of Charles’ injury?Was it foreseeable that the sheriff’s failure to question Allen on his psychiatric history would result in the injury to Charles?What is sovereign immunity, and will it offer any protection to the sheriff?Name your file SUO_LGS1004_W2_A2_Freres_J.doc
South University Actions Of The Florida State Sheriff Memorandum

AIU Principles of Marketing Pricing Discussion.

In your own words, answer this unit’s discussion questions in a main post (recommended minimum 200 words), and respond to at least 2 peers’ postings (recommended minimum 75 words).After you have reviewed the Assignment Details below, click the Discussion Board link under the My Work heading above to open the Discussion Board and make your post.Use these videos for help on how to post to the Discussion Board:AIU Student Mobile app viewDesktop viewAssignment DetailsCompanies adopt various pricing strategies that appeal to their target market. Marketers must also balance the price that generates the required profits for the company to operate successfully.Describe an example of a pricing strategy that affected a purchase decision that you made.Responses to PeersRead through your peers’ posts, and respond to a minimum of 2 of your peers. You can connect to your peers in several ways, including the following:Explain how the purchase decision that you described in your post is similar to or different from those of your peers.In your own words, please post a response to the Discussion Board and comment on other postings. You will be graded on the quality of your postings.For assistance with your assignment, please use your text, Web resources, and all course materials.Discussion Board Reminders: Must have three posts: A Main Post and two replies to peers.First post: Either your main post or a reply to others must be posted before midnight CT (Central time) on Friday of each week.Second and third post: Must be posted on a different day from the first post.Connect to Content: At least one post must refer to course learning materials. See the Academic Resource on Discussion Boards for help with connecting to the content.Engaging in Class Discussions: For more information on making the most of your class dialog, review the Academic Resource on Discussion Boards. Grading RubricAssignment CriteriaProficient DescriptorPointsQuality of Main Post18 Points TotalStudent’s main post meets or exceeds the following requirements:Responds completely to all of parts of discussion question (5 points)Communicates content information accurately and/or logically (5 points)Delivers a thoughtful response demonstrating insights and reflections (4 points)Connects to both key content concepts and personal experiences (4 points)/18pts.Response to Peers16 PointsTotalResponds to a minimum of two peers (8 points per response)Substantive response (4 points)Furthers the conversation with peers. Examples could include (4 points):Provides additional resourcesConnects to key conceptsShares personal or professional experiencesContributes new information or perspectivePresents a competing viewpoint/16 pts.Support from Learning Resources3 PointsTotalAt least one post refers to course learning resources./3 pts.Professional Writing3 pointsResponse is well-organized, clear, and free of grammatical and mechanical errors.Posts demonstrate courtesy and respect for others./3 pts.Total40 points possible/40 pts.
AIU Principles of Marketing Pricing Discussion

Urinalysis of Four Urine Samples

Urinalysis of Four Urine Samples. Urinalysis Practical Background: As you have learned, the urinary system performs many vital functions in the body including: Regulating blood volume and pressure by regulating water excretion, Regulating plasma ion/solute concentrations by adjusting urine composition, Assisting blood pH stabilisation, Removing nitrogenous waste, Conserving water and important nutrients and Assisting the liver in detoxifying poisons. Therefore, analysing a sample of urine from a person can provide important information on the health of that person. Urinalysis can reveal diseases such as diabetes mellitus, urinary tract infections and renal (kidney) infections such as glomeronephritis and kidney stones (renal calculi). A medical professional may perform a urinalysis for several reasons: As a general health check-up, Diagnosing metabolic or systemic diseases that affect renal function (heart failure will lead to decreased blood flow to the kidneys, pre-eclampsia during pregnancy will lead to increased protein in the urine), Diagnosis of endocrine disorders e.g. infertility (low levels of FSH and/or LH), Diagnosis of urinary system disease, Monitoring of glucose levels in patients with diabetes, Testing for pregnancy (hCG levels secreted by the embryo), Screening for drug use. Urinalysis is a technique involving physical, chemical and microscopic analyses of a sample of urine. Physical parameters: Normal urine is a clear yellow colour due to the presence of uribilin. Abnormal urine may be dark orange, red or brown and cloudy in appearance. This can be due to the presence of red and/or white blood cells or pigments and may indicate a urinary tract or renal infection or disease, liver or gall bladder disease. Normal urine has a specific gravity of between 1.002 – 1.028 (this is a measure of the number of particles/solutes in the urine, its concentration). A urine sample that has an elevated specific gravity can indicate dehydration, diarrhea/vomiting, glucosuria, inappropriate ADH secretion. A diminished specific gravity may indicate such diseases as renal failure or pyelonephritis. Chemical parameters: The chemical analysis of urine is routinely performed using an inexpensive and relatively accurate dipstick test (Uristix from Bayer or other brands). The test uses a reagent-coated plastic stick that is placed or dipped into the urine sample. The reagent areas change colour according to the presence of glucose and/or protein. (a) Figure 1. Colour chart (a) for determination of glucose and/or protein. The glucose test on the dipstick is based on a double sequential enzyme reaction. One enzyme, glucose oxidase, catalyses the formation of gluconic acid and hydrogen peroxide from the oxidation of glucose (if present in the urine). A second enzyme, peroxidase, catalyses the reaction of hydrogen peroxide with a potassium iodide chromogen to oxidise the chromogen to colours ranging from green to brown. Normal urine has less than 0.1% glucose concentration. The protein test on the dipstick is based on the protein-error-of-indicators principle. At a constant pH, the development of any green colour is due to the presence of protein. Colours range from yellow for ‘negative’ through yellow-green and green to green-blue for ‘positive’ reactions. Normal urine has a protein concentration of less than 100 µg/ml. Although the dipstick test is semi-quantitative, significantly more accurate levels of glucose and protein can be determined by other means. In this practical you will use a BCA Assay (discussed later) to quantify the amount of protein present in a sample of urine. Urinalysis may also include assaying for levels of ketones (an indicator of diabetic ketosis, fasting or starvation), blood cells (indicating infection or kidney stones), bilirubin (liver or gall bladder disease), drugs and many other substances. Microscopic parameters: The urine sample can also be analysed by a microscope, often after staining to reveal any pathogens such as bacteria, urine crystals, cells and/or mucous. The presence of any of these may indicate infection or disease and further medical investigation will provide a thorough diagnosis. Aim: The aim of this practical is to perform glucose and protein urinalysis techniques on five samples of ‘urine’ provided by five ‘patients’ and use this information to provide an initial diagnosis for each patient. Part One: Using Dipsticks To Provide A Qualitative Measure of Protein And/Or Glucose. Materials: 5 samples of urine labelled A – E (these will be required for Parts One and Two), 5 Uristix dipsticks. Method: Perform a basic physical analysis of the urine samples noting the colour and cloudiness of each sample: Urine A Urine B Urine C Urine D Urine E Colour Cloudiness Immerse a dipstick into each of the samples, wait 60 seconds and record your results using the colour chart in Figure 1 to determine if the sample contains glucose and/or protein or neither substance: Urine A Urine B Urine C Urine D Urine E Glucose Protein Ketones Negative Negative Negative Negative Strongly positive Blood Negative Negative Trace Negative Negative Part Two: Using A Commercial BCA Assay To Provide A Quantitative Measure of Protein. Background: The BCA Protein Assay exploits the chemical reduction of Cu2 to Cu1 by protein in an alkaline medium with the selective colorimetric detection of the cuprous cation (Cu1 ) by bicinchoninic acid (BCA). The first step is the chelation of copper with protein in an alkaline environment to form a blue coloured complex. In this reaction, known as the biuret reaction, peptides containing three or more amino acid residues form a coloured chelate complex with cupric ions in an alkaline environment containing sodium potassium tartrate. Single amino acids and dipeptides do not give the biuret reaction, but tripeptides and larger polypeptides or proteins will react to produce the light blue to violet complex that absorbs light at 540 nm. In the second step of the colour development reaction, BCA, a highly sensitive and selective colorimetric detection reagent reacts with the Cu1 that was formed in step 1. The purple-coloured reaction product is formed by the chelation of two molecules of BCA with one Cu1 . The BCA/Cu complex is water-soluble and exhibits a strong linear absorbance at 562 nm with increasing protein concentrations. The rate of BCA colour formation is dependent on the incubation temperature, the types of protein present in the sample and the relative amounts of reactive amino acids contained in the proteins. Figure 2. Reaction diagram for the bicinchoninic acid (BCA) protein assay. Materials: The 2 samples of urine from Part One that were positive for protein, Protein stock standard (BSA, bovine serum albumin) at 1mg/ml, 0.9% Saline (diluent) BCA (bicinchoninic acid) Working Reagent (labelled BCA WR), 6 Tubes for dilutions for the standard curve, 96 Well microtitre plate, P100, P200Urinalysis of Four Urine Samples

Northern Virginia Business Continuity & Disaster Recovery in Cloud Computing Discussion

help me with my homework Northern Virginia Business Continuity & Disaster Recovery in Cloud Computing Discussion.

Business Continuity and Disaster Recovery in Cloud Computing In many organizations, the primary role of the IT staff is to ensure 99.999% availability of computing resource applications, power, files, networks, and phone systems to name a few.In this assignment, imagine that you have been hired by the midsized, multinational, BIG-CLOUD company to create and graphically depict a cloud based business continuity plan (BCP) and disaster recovery plan (DRP). Your solution must include details on how the cloud solution of your choice will address three (3) or more high fault items such as user disk failure, server disk failure, network failure, database failure, phone system failure, server power failure, desktop power failure, fire, flood, and malware. (Use the template in Chapter 10 of the text as a reference for the BCP and DRP plan.) Fictitious assumptions and details may be assumed or created for the completion of this assignment.Write a five to eight (5-8) page paper in which you:Provide a brief background on the company that has hired you and describe its backup and recovery challenges.Describe the threats to its IT data center infrastructure.Create a BCP plan for the high fault items that are being addressed.Create a DRP plan for the high fault items that are being addressed.Create a section in your BCP that addresses the mitigation strategy of your cloud solution in regard to the high fault items listed in the scenario.Create a section in your DRP that addresses the mitigation strategy of your cloud solution in regard to the high fault items listed in the scenario.Discuss the amount of control your organization wants to retain over its data, in relation to moving information to a third party, and data leaving the country or region boundaries. Assume both situations are such that allows for a cloud-based backup solution.Evaluate the pros and cons of your cloud-based backup solution with regards to your organization’s requirements (i.e., flexibilities you gain versus absolute control you lose). Determine whether the pros outweigh the cons.Use at least three (3) quality resources in this assignment. Note: Wikipedia and similar Websites do not qualify as quality resources.Your assignment must follow these formatting requirements:Be typed, double spaced, using Times New Roman font (size 12), with one-inch margins on all sides; citations and references must follow APA or school-specific format. Check with your professor for any additional instructions.Include a cover page containing the title of the assignment, the student’s name, the professor’s name, the course title, and the date. The cover page and the reference page are not included in the required assignment page length.
Northern Virginia Business Continuity & Disaster Recovery in Cloud Computing Discussion

CMIT 320 University of Maryland University College HIPPA and PHI Privacy Discussion

CMIT 320 University of Maryland University College HIPPA and PHI Privacy Discussion.

Hello,I would like this Assignment done in Microsoft Powerpoint. Requirements- 8 total slides. 2 slides per topic Topics and instructions are as follows:The human resource department is updating its HIPAA Basic Training for Privacy and Security course. As a security analyst for the hospital, you have been tasked with covering the topics in the training related to the HIPAA security rule and the information that hospital staff need to know regarding personally identifiable information (PII), personal health information (PHI), and electronic personal health information (ePHI) to comply with federal regulations.You will submit your presentation. Include two slides for each bullet below (minimum 8 slides total) explaining the following:HIPAA Security RuleHIPAA, PII, PHI, and ePHI DefinitionsSafeguarding of PII, PHI, and ePHIDisclosures of PII, PHI, and ePHII am also attaching a Word document with all the instructions as well. Make sure to use all the concepts listed above in the powerpoint. I would like this to be original work.
CMIT 320 University of Maryland University College HIPPA and PHI Privacy Discussion


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